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M94A0581.TXT
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1994-10-21
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Document 0581
DOCN M94A0581
TI Pseudopancreatitis in HIV disease.
DT 9412
AU Parkin D; Edwards R; Department of HIV Medicine, Royal North Shore
Hospital, St; Leonards, NSW.
SO Annu Conf Australas Soc HIV Med. 1993 Oct 28-30;5:96 (poster no. 43).
Unique Identifier : AIDSLINE ASHM5/94349074
AB We report a case of hyperamylasemia in a 39 year old AIDS patient. He
presented in May 1992 with Pneumocystis carinii pneumonia and was
subsequently found to be HIV antibody positive. His CD4 Count was 32. On
recovery AZT 600mg a day and Bactrim were commenced. In November 1992
CMV retinitis was diagnosed and Ganciclovir was commenced.
Antiretroviral therapy was changed to ddl in November 1992. During April
1993 the patient complained of increasing abdominal discomfort and
nausea. From February to April 1993 a slowly progressive rise in serum
amylase was noted, attributed to pancreatitis, and ddl was ceased. In
May 1993 the patient complained of xerostomia and bilateral parotid
swelling was noted. The diagnosis of bilateral parotitis was made. A
salivary gland nuclear medicine study confirmed bilateral parotid
enlargement. Fractionation of the amylase in early May revealed 85% from
the salivary glands, 15% from the pancreas. With conservative management
the parotitis resolved and the serum amylase returned to near normal
levels. This case serves to highlight that salivary gland dysfunction
should be considered as a cause of hyperamylasemia in HIV/AIDS patients.
DE Acquired Immunodeficiency Syndrome/*DRUG THERAPY/ENZYMOLOGY Adult
Amylases/*BLOOD AIDS-Related Opportunistic Infections/*DRUG
THERAPY/ENZYMOLOGY Case Report Diagnosis, Differential Human Male
Pancreatitis/*CHEMICALLY INDUCED/ENZYMOLOGY Parotitis/*CHEMICALLY
INDUCED/ENZYMOLOGY Saliva/ENZYMOLOGY MEETING ABSTRACT
SOURCE: National Library of Medicine. NOTICE: This material may be
protected by Copyright Law (Title 17, U.S.Code).